Posted by rozeklawoffice on January 14, 2012 under Carbon Monoxide Poisoning, General | 
Green Bay Hilton Garden Inn
As news surfaced of yet another carbon monoxide poisoning at a Green Bay hotel, it shed light on the reluctance of the hotel industry to address a major safety concern. On December 30, 2011, as many as 16 people were hospitalized after being exposed to dangerous levels of carbon monoxide while staying at the Green Bay Hilton Garden, 1015 Lombardi Ave., Green Bay, Wisconsin. The Green Bay Fire Department recently released their records that indicate four small children and several other people were suffering from common symptoms of carbon monoxide exposure, including vomiting, nausea and headaches.
The Green Bay Fire Department detected dangerous levels of carbon monoxide in several areas within the hotel, including the swimming pool area, the workout room, two restrooms and a stairway. Apparently the carbon monoxide level in the swimming pool area was a 800 parts per million (ppm) and 957 ppm in the pool equipment room. Buildings are typically evacuated at 9 ppm. Sustained exposure to carbon monoxide at levels as high as 800 ppm can be lethal. Carbon monoxide exposure can also result in severe long-term consequences, such as brain injury and heart problems. The Hilton Garden Inn general manager, Michelle Lang, has continued to remain silent as to how this could have happened to their guests.
Green Bay Hilton Garden Inn Swimming Pool
The hotel industry has long recognized the dangers of carbon monoxide exposure, yet some hotel chains have failed to institute uniform policies requiring the installation of carbon monoxide detectors throughout their hotels. Ironically, exactly one year prior to the date of the Green Bay Hilton Inn carbon monoxide poisoning incident, our office joined forces with the Brain Injury Law Group to file suit on behalf of victims who were poisoned by carbon monoxide while staying at the Green Bay Day’s Inn back in May of 2009. Now 2 1/2 years later, it is apparent that some hotel chains have still done nothing to protect their guests from carbon monoxide exposure, like simply installing working carbon monoxide detectors. Under Wisconsin Safe Place Law, hotel owners are required to keep their hotels as “free from danger to the life, health, safety, and welfare of guests as the nature of the hotel will reasonably permit.” The failure to install carbon monoxide detectors throughout a hotel clearly violates this law and until the hotel industry gets this message loud and clear, hotel guests throughout the world will continue to get sick and even die from carbon monoxide poisoning.
Posted by rozeklawoffice on December 11, 2011 under General, Mild Brain Injury, Traumatic Brain Injury |
Traumatic Brain Injuries can lead to many long term health problems. Early cognitive decline is a leading long term consequence of TBI, which can ultimately lead to Chronic Traumatic Encephalopathy, Alzheimer’s Disease, Parkinson’s Disease, and early onset Dementia. Autopsy studies of professional athletes have been able to confirm that multiple concussions or brain injuries can result in a condition called Chronic Traumatic Encephalopathy.
Chronic Traumatic Encephalopathy is a progressive disease that results in a gradual decline in cognitive function. Victims of Chronic Traumatic Encephalopathy exhibit symptoms consistent with dementia, including confusion, memory loss, depression, and aggressive behavior. The hallmark indicator of Chronic Traumatic Encephalopathy on autopsy of the brain was the buildup of a plaque referred to as tau protein. While it was always understand that repeated concussions could result in the buildup of tau protein, it wasn’t until recently that researchers were able to identify the buildup of tau protein in individuals with only a single head injury. (Victoria E. Johnson, William Stewart, Douglas H. Smith. Widespread Tau and Amyloid-Beta Pathology Many Years After a Single Traumatic Brain Injury in Humans. Brain Pathology, 2011; DOI: 10.1111/j.1750-3639.2011.00513.x).
Alzheimer’s Disease is a progressive form of dementia. It is the most common type of dementia, resulting in confusion, anger outbursts, lack of long-term memory, and language deficits. The cost of Alzhiemer’s Disease on society is great. Because most Alzheimer’s patients will continually and progressively decline, at some point they will have to rely upon others for their care. While family members and loved ones usually try to provide care initially, the challenges of caring for an Alzheimer’s sufferer can take an incredible toll on every aspect of a caregiver’s life. What makes it additionally challenging and unrewarding is that, at some point, the Alzheimer’s sufferer will no longer have the long-term memory required to remember the identity of their caregiver. On autopsy it has been show that tau protein buildup is also a hallmark indicator of patients with Alzheimer’s Disease. Studies have shown that brain injury victims have an increased risk of developing Alzheimer’s Disease.
Parkinson’s Disease is a progressive disorder of the central nervous system with its origins in the midbrain. The initial symptoms of Parkinson’s Disease involve movement disorders, but later Parkinson’s Disease patients typically develop cognitive symptoms including dementia. Parkinson’s Disease, while typically idiopathic in origin, can follow traumatic brain injury that results in damage to the basal ganglia which can result in a dopamine production disruption. (Traumatic Brain Injury in Adult Rats Causes Progressive Nigrostriatal Dopaminergic Cell Loss and Enhanced Vulnerability to the Pesticide Paraquat Che Brown Hutson, Carlos R. Lazo, Farzad Mortazavi, Christopher C. Giza, David Hovda, and Marie-Francoise Chesselet Journal of Neurotrauma 2011 28:9, 1783-1801). Unfortunately, there is no lab test to definitively diagnose Parkinson’s Disease as of yet. Due to the degenerative nature of Parkinson’s Disease, patients and their loved ones face an enormous burden when dealing with long-term care for the Parkinson’s patient.
As if traumatic brain injury survivors did not have enough to deal with just attempting to recover from their injury, now studies have established that victims of TBI have an increased risk of developing early onset dementia, Chronic Traumatic Encephalopathy, Alzheimer’s Disease, and Parkinson’s Disease.
Posted by rozeklawoffice on October 25, 2011 under Traumatic Brain Injury |
New types of brain imaging techniques may be able to help traumatic brain injury survivors in identifying the exact nature, extent and location of their brain trauma. The most promising imaging technique involves an advanced MRI protocol referred to as SWI.
Susceptibility Weighted Imaging (SWI) is a relatively new imaging technique that can be performed on most General Electric and Siemens MRI machines (1.0 T, 1.5 T, 2.0 T & 3.0 T). SWI can be included in the ordinary brain injury scan protocol. SWI images have been described as being 5 times as clear as ordinary MRI. SWI is particularly sensitive to the detection of iron deposits in the brain.Whenever there is a bleed in the brain, iron is deposited and remains, even after the liquid is reabsorbed into the brain. MRI and SWI can identify these iron deposits as lesions.
The majority of closed head injuries that result in brain damage results in a Diffuse Axonal Injury (DAI), which results in relatively small lesions throughout the brain. DAI lesions typically occur at the gray-white matter interface in the brain. This is where gray matter meets white matter. Due to the differences in density of gray matter vs. white matter, brain axons at this gray-white matter interface are more susceptible to damage from trauma than axons in other areas of the brain. SWI can more easily identify gray-white matter interface lesions than other MRI protocols. The following image compares a typical MRI image to that of an SWI image depicting lesions:
Standard 1.5 T MRI Image (Left) vs. SWI Image (Right)
The clarity of the SWI image compared to that of the standard MRI image is remarkable. The benefits of SWI for TBI survivors are many. All TBI survivors will benefit by their doctors being able to pinpoint the areas of the brain that are the most damaged. Healthcare providers can tailor their treatment program to focus more treatment and rehabilitation on those areas of the brain that have the most damage. Also, for victims of mild TBI, SWI may finally be the key to establishing the objective proof of injury. Mild TBI survivors have long been frustrated by the lack of any objective evidence of injury, despite the dramatic changes in the TBI survivors’ lives and ability to function. SWI can help identify the exact nature, extent and location of the mild TBI survivors’ brain damage, which will lead to better care, treatment and rehabilitation for the mild TBI survivor.
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If you or a loved one have sustained a TBI and need an experienced Wisconsin Brain Injury Attorney, contact Rozek Law Offices for a free initial consultation.
Posted by rozeklawoffice on October 21, 2011 under General |
Rozek Law Offices is pleased to announce that we have opened a Madison office as of October 2011. The Madison office was opened primarily for the convenience of our current and future clients, as we represent personal injury victims in Dane County and throughout the state.
The new office is conveniently located on Madison’s East Side in the Park Bank Plaza building near I-94 and Highway 151.
If you or a loved one are in need of any of the following, please do not hesitate to contact our office:
The new office information is as follows:
Rozek Law Offices, SC
2810 Crossroads Drive
Suite 4000
Madison, Wisconsin 53718
Telephone: 608/208-1147
